ED, No Libido, Seeing A Urologist Now

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@Stpfan Your TSH is moderately elevated and free T3 & free T4 on the low side. I've been focusing on my thyroid function, self treating with desiccated thyroid, along with rx t-shots. (25 mg, IM, 3X week) My libido is improving(sexual thoughts, desire) and slight improvement in erectile function(more nocturnal erections).

I'm not the first one on this forum to recommend dealing with the thyroid. It's the spark plug of the body.
 
Defy Medical TRT clinic doctor
@Stpfan Your TSH is moderately elevated and free T3 & free T4 on the low side. I've been focusing on my thyroid function, self treating with desiccated thyroid, along with rx t-shots. (25 mg, IM, 3X week) My libido is improving(sexual thoughts, desire) and slight improvement in erectile function(more nocturnal erections).

I'm not the first one on this forum to recommend dealing with the thyroid. It's the spark plug of the body.

I appreciate the post. I constantly tell my Endo to adjust my thyroid and/or look over the lab work. He refuses... he says everything is dialed in to his liking. I will make another comment when I see him or talk with my PCP.
 
I wanted to give another update to my new protocol. This is odd to me.... didn't expect this. How does your Hemoglobin levels decrease on their own??? Like... drastically change?

Currently I'm taking

Testosterone Cyp 80mg (every 4 days)
Deca 45mg (every 4 days)
HCG 300IU (every 4 days)
Anastrozole 0.25mg (every 4 days)

My old protocol... I was only taking Test Cyp 65-70mg (every 4 days) with nothing else... here are a list of Hemoglobin levels from the Red Cross...

08/22/2019 - 18.4
01/08/2020 - 19.2
03/04/2020 - 18.7
05/04/2020 - 17.6
06/29/2020 - 18.1
08/24/2020 - 17.4
10/19/2020 - 19.4
12/14/2020 - 17.6
02/08/2021 - 18.2
04/05/2021 - 13.8

I've been adding Deca, Anastrozole, and HCG and how in the heck did my Hemoglobin drastically reduce? I asked the Red Cross to recheck. They said we only recheck when your Hemoglobin is 12.9 or lower. We also don't check a 2nd time if your Hemoglobin is 20.0 or higher. I was in shock. What happened? I donated anyways.
 
STPFAN are you able to give us an update on how you are feeling in terms of libido? Reading through the posts you mentioned you are feeling 100% better? If this is true, what do feel was the game changer? Was it the AI, adding Nandrolone or both? How are you feeling right now? Did you ever look into DHT?
 
Does anyone remember the supplement called 6-OXO that was available from approximately 2007 to 2008? It was a supplement marketed towards fitness/exercise and I believe it was supposed to be an aromatase inhibitor. It put my libido through the roof. Unfortunately it was banned by the FDA, because I believe they discovered that it did increase Testosterone levels or had some other affect on the endocrine system. I wish I had brought several cases of that supplement when it was available. If anyone else tried it, did it increase libido for you?
 
At MADMAN (or whoever would like to chime in)

I just have a quick question without starting an entirely different thread from one "off topic" question.

For celebrities such as Dwayne The Rock Johnson, Triple H, John Cena, and Arnold Schwarzenegger... we all know their Test Cyp... and other steroids are off the charts... In your opinion... how do they manage their High Hematocrit? Are they donating blood every 30 Days? 40 Days? 60 Days? In your opinion what would be their routine? Are they constantly taking Iron supplements after a quick 30 day donation?

Don’t stress about HGB and HCT too much, people worry way too much about these levels. Dr Neil Rouzier has said he’s had his HCT levels close to 60 for around 20 years without any issues. And I just saw Dr Jordan Grant, who is a very intelligent eurologist, I believe, that is well known in a few TRT fb groups, say that his HCT is around 57 without any issues.

Where do ur platelets sit? As long as they’re not too high, don’t stress HGB and HCT levels too much. Let them fall where they fall. And if u feel the need to donate every so often, do so, but don’t feel the need to donate very often and risk depleting ur iron levels. Here’s a quick video where Dr Neil Rouzier sums up his opinion on the issue

 
@Stpfan

I would not be too concerned with running higher-end levels (low 50s) if you are not experiencing any symptoms let alone feel well overall.

Once levels approach 54-55% most would recommend addressing the issue whether through blood donations or in many cases lowering the dose of T.

Need to keep in mind where your hematocrit sat pre-trt.

Some men are naturally on the higher-end pre-trt whereas many others fall well within range and there are also the ones with borderline/low levels due to underlying issues/anemia.



Come to your own conclusion!


--------------------------------------------------------------------------------------------------
EVALUATION AND MANAGEMENT OF TESTOSTERONE DEFICIENCY: AUA GUIDELINE (2018)


Adjunctive Testing


6. In patients with low testosterone, clinicians should measure serum luteinizing hormone levels. (Strong Recommendation; Evidence Level: Grade A)

7. Serum prolactin levels should be measured in patients with low testosterone levels combined with low or low/ normal luteinizing hormone levels. (Strong Recommendation; Evidence Level: Grade A)

8. Patients with persistently high prolactin levels of unknown etiology should undergo evaluation for endocrine disorders. (Strong Recommendation; Evidence Level: Grade A)

9. Serum estradiol should be measured in testosterone deficient patients who present with breast symptoms or gynecomastia prior to the commencement of testosterone therapy. (Expert Opinion)

10. Men with testosterone deficiency who are interested in fertility should have a reproductive health evaluation performed prior to treatment. (Moderate Recommendation; Evidence Level: Grade B)

11. Prior to offering testosterone therapy, clinicians should measure hemoglobin and hematocrit and inform patients regarding the increased risk of polycythemia. (Strong Recommendation; Evidence Level: Grade A)

12. PSA should be measured in men over 40 years of age prior to commencement of testosterone therapy to exclude a prostate cancer diagnosis. (Clinical Principle)




GUIDELINE STATEMENTS

11. Prior to offering testosterone therapy, clinicians should measure hemoglobin and hematocrit and inform patients regarding the increased risk of polycythemia. (Strong Recommendation; Evidence Level: Grade A)

Polycythemia, sometimes called erythrocytosis, is generally defined as a hematocrit (Hct) >52%. It is categorized into primary (life-long), often related to genetic disorders; and secondary (acquired), which is attributed to polycythemia vera, living at high altitude, hypoxia (e.g., chronic obstructive pulmonary disease, obstructive sleep apnea, tobacco use), paraneoplastic syndromes, and testosterone therapy.187 188

Prior to commencing testosterone therapy, all patients should undergo a baseline measurement of Hb/Hct (Appendix C). If the Hct exceeds 50%, clinicians should consider withholding testosterone therapy until the etiology of the high Hct is explained.187
While on testosterone therapy, a Hct ≥54% warrants intervention. In men with elevated Hct and high on-treatment testosterone levels, dose adjustment should be attempted as first-line management. In men with elevated Hct and low/normal on-treatment testosterone levels, measuring an SHBG level and a free testosterone level using a reliable assay is suggested. If SHBG levels are low/free testosterone levels are high, dose adjustment of the testosterone therapy should be considered. Finally, men with elevated Hct and on treatment low/normal total and free testosterone levels should be referred to a hematologist for further evaluation and possible coordination of phlebotomy.

Androgens have a stimulating effect on erythropoiesis and elevation of Hb/Hct is the most frequent adverse event related to testosterone therapy.189-191 During testosterone therapy, levels of Hb/Hct generally rise for the first six months and then tend to plateau.192, 193

Among 5 randomized, placebo-controlled trials that evaluated Hb and Hct levels in men (mean baseline testosterone <300 ng/dL) using either gel, solution, or IM testosterone therapy for 12 weeks to 1 year, a significant increase in the incidence of elevated Hct was observed in those using testosterone (OR=6.46; CI: 1.86, 22.40) compared to those on placebo.194-201 The calculated odds ratio belies the number of polycythemia events in absolute terms; 19 events in 1,094 patients occurred in the treatment arm as compared to 1 event in 1,093 patients in the placebo group.

While the incidence of polycythemia for one particular modality of testosterone compared to another cannot be determined, trials have indicated that injectable testosterone is associated with the greatest treatment-induced increases in Hb/Hct. In the current meta-analysis of RCTs, long-acting IM testosterone resulted in a mean increase in Hb levels of 1.4 mg/dL compared to 1.6 mg/dL with short-acting IM testosterone, 0.9 mg/dL with transdermal preparations, and 0.7 mg/dL with topical patches.150, 182, 194-196, 201-218 This is likely due to the high and sometimes supra-physiological levels of testosterone that occur in the early days after injection.219 A retrospective comparative series involving 175 men with testosterone deficiency who used different modalities of testosterone therapy reported that 19% of men receiving IM testosterone experienced polycythemia compared to 12.5% with testosterone pellets and 5.4% with gels.220

It is unclear if the risk of polycythemia is greater in men with comorbid disorders that predispose to hypoxia, such as chronic obstructive pulmonary disease or obstructive sleep apnea.188 It is also currently unknown if rates of polycythemia are associated only with short-acting injectable agents or occur with equal frequency when using the longer-acting testosterone undecanoate.221

--------------------------------------------------------------------------------------------------


The use of exogenous T will result in driving up RBCs/hemoglobin/hematocrit within the first month of starting trt and can take up to 9-12 months to reach peak levels.

In many cases, levels can stabilize in due time but many will also struggle with high levels and try to manage with frequent blood donations.

In most cases running too high TT/FT levels will result in high hematocrit.

My reply from another thread:Daily Injection Experiment

Injectable T has been shown to have a greater impact on increasing HCT compared to transdermal T.

3–18% with transdermal administration and up to 44% with injection.

In most cases when using injectable T high supra-physiological peaks post-injection and overall T levels (running too high TT/FT level) will have a big impact on increasing HCT.

Manipulating injection frequency by injecting more frequently using lower doses of T resulting in minimizing the peak--->trough and maintaining more stable levels may lessen the impact on HCT but it is not a given.

As again running very high FT levels will have a stronger impact on driving up HCT.

T formulation, the dose of T, genetics (polymorphism of the AR), age all play a role in the impact a trt protocol will have on blood markers (RBCs/hemoglobin/hematocrit).

Other factors such as sleep apnea, smoking can have a negative impact on hematocrit.
 
Again... just to update my current situation. I donated blood on Monday April 5, 2021. Upon checking of my Hemoglobin... it was awfully low. The Red Cross claims it was at 13.8 I quickly made an appointment to my back up Endocrinologist to write some orders for some labs. I needed to know what happened? Why was the Red Cross system off? Or was it really accurate? Didn't make any sense. Here are more newest labs after donating blood.

Labs From March 9, 2021

WBC Count 7.1 K/uL
(Standard Range 4.0 -11.3)
RBC Count 5.71 M/uL (Standard Range 4.30 - 5.90)
Hemoglobin 15.8 g/dL (Standard Range 13.6 -17.6)
*LOWEST HEMOGLOBIN EVER* WOW
Hematocrit 48.3% (Standard Range 40.0 - 50.0%)
*LOWEST HEMATOCRIT EVER* WOW
MCV 84.5 fl (Standard Range 82 - 97)
MCH 27.7 pg (Standard Range 27.3 - 33.4)
MCHC 32.8 g/dL (Standard Range 32 - 36)
RDW 14.4% (Standard Range 11.6 - 14.8)
Platelet Count 269 K/uL (Standard Range 150 - 450)
Mean Platelet Volume 9.2 fL (Standard Range 7.4 - 10.4 fL)

Sodium 141 mmol/L (Standard Range 135 - 145 mmol/L)
Potassium 4.9 mmol/L (Standard Range 3.5 - 5.2 mmol/L)
Chloride 107 mmol/L (Standard Range 98 - 111 mmol/L)
Total CO2 26 mmol/L (Standard Range 20 - 29 mmol/L)
Anion Gap 8 mmol/L (Standard Range 5 - 20 mmol/L)
Glucose 100 mg/dL (Standard Range 60 - 99 mg/dL)
Blood Urea Nitrogen 34 mg/dL (Standard Range 7 - 25 mg/dL)
Creatinine 1.04 mg/dL (Standard Range 0.60 - 1.30 mg/dL)
Glomerular Filtration Rate 88 mL/min/1.73 m2 (Standard Range >60mL)
*Best Filtration Ever On Record* WOW
Calcium 8.8 mg/dL (Standard Range 8.5 - 10.5 mg/dL)
Protein 6.5 g/dL (Standard Range 6.4 - 8.3 g/dL)
Albumin 4.2 gm/dL (Standard Range 3.5 - 5.1 gm/dL)
Bilirubin 0.9 mg/dL (Standard Range 0.3 - 1.2 mg/dL)
AST (SGOT) 33 U/L (Standard Range <35 U/L)
ALT (SGPT) 38 U/L (Standard Range 9 - 47 U/L)
Alkaline Phosphate 60 U/L (Standard Range 33 - 120 U/L)

TSH 4.18 ulU/mL (Standard Range 0.4 - 4.5 ulU/mL)
Free T4 1.00 ng/dL (Standard Range 1.7 - 3.7 ng/dL)

Total Testosterone >1500 (Standard Range 240 - 890 ng/dL)
My doctor only orders the lab in which once past 1500 it doesn't calculate. I couldn't change the order.

PSA Diagnostic 1.0 ng/mL (Standard Range 0.0 - 4.o ng/mL)

It's still mind boggling that I have the lowest RBC, Hemoglobin, Hematocrit since adding Deca and HCG. It blows my mind. I asked my 2nd Endocrinologist what he thought.... and he just said it doesn't make much sense either and I DO NOT ADVISE you taking Deca, HCG on your current protocol. But, I understand you want to live life differently than most people.
 
STPFAN are you able to give us an update on how you are feeling in terms of libido? Reading through the posts you mentioned you are feeling 100% better? If this is true, what do feel was the game changer? Was it the AI, adding Nandrolone or both? How are you feeling right now? Did you ever look into DHT?

Thanks for the post. I'm trying to take 200mg of Test Cyp, 100mg of Deca, HCG around 600 IU A Week, and Anastrozole 0.25mg every 4 days. I don't want to drop my Estrogen so low... I'm going to have to dial in it better. So what I'm doing is this at the moment...

Test Cyp 85mg
Deca 42.5mg
HCG 257 IU
Every 3 Days

Anastrozole 0.25mg (Every 4 days) until I can get more accurate lab work drawn up.

The funny thing is I screwed up injecting the other day... I was in a rush. I did a 1 to 1 ratio with Test Cyp and Deca... I honest to God had the best sex in a LONG LONG TIME. I didn't lose my erection at all... I actually started feeling sensations that I haven't in quite some time. I think I'm losing my erection because I just don't feel anything! Like nothing! That's my opinion... but on this new protocol... with HCG... it's doing something different. It's just enough sensation right now to keep the erection going without losing it. And it's pretty strong. I just hope it continues... I didn't need to use Viagra or any herbal supplement. The exact dosage that made this work was... Test Cyp 42.5mg, Deca 42.5mg, HCG 257 IU, Anastrozole 0.25mg. I took it all at once. And yes I did screw up... I want to go with a 2 to 1 ratio with Test Cyp to Deca. But like I said... maybe it was a good thing that I did screw up? Might of found some magic? I will keep you posted.

The Deca does seem to be increasing my blood pressure? And I'm around 100mg a week now. At the Red Cross it came in at 150/62.
 
Don’t stress about HGB and HCT too much, people worry way too much about these levels. Dr Neil Rouzier has said he’s had his HCT levels close to 60 for around 20 years without any issues. And I just saw Dr Jordan Grant, who is a very intelligent eurologist, I believe, that is well known in a few TRT fb groups, say that his HCT is around 57 without any issues.

Where do ur platelets sit? As long as they’re not too high, don’t stress HGB and HCT levels too much. Let them fall where they fall. And if u feel the need to donate every so often, do so, but don’t feel the need to donate very often and risk depleting ur iron levels. Here’s a quick video where Dr Neil Rouzier sums up his opinion on the issue


I appreciate the post and information!! Thanks!
 
@Stpfan

I would not be too concerned with running higher-end levels (low 50s) if you are not experiencing any symptoms let alone feel well overall.

Once levels approach 54-55% most would recommend addressing the issue whether through blood donations or in many cases lowering the dose of T.

Need to keep in mind where your hematocrit sat pre-trt.

Some men are naturally on the higher-end pre-trt whereas many others fall well within range and there are also the ones with borderline/low levels due to underlying issues/anemia.



Come to your own conclusion!


--------------------------------------------------------------------------------------------------
EVALUATION AND MANAGEMENT OF TESTOSTERONE DEFICIENCY: AUA GUIDELINE (2018)


Adjunctive Testing


6. In patients with low testosterone, clinicians should measure serum luteinizing hormone levels. (Strong Recommendation; Evidence Level: Grade A)

7. Serum prolactin levels should be measured in patients with low testosterone levels combined with low or low/ normal luteinizing hormone levels. (Strong Recommendation; Evidence Level: Grade A)

8. Patients with persistently high prolactin levels of unknown etiology should undergo evaluation for endocrine disorders. (Strong Recommendation; Evidence Level: Grade A)

9. Serum estradiol should be measured in testosterone deficient patients who present with breast symptoms or gynecomastia prior to the commencement of testosterone therapy. (Expert Opinion)

10. Men with testosterone deficiency who are interested in fertility should have a reproductive health evaluation performed prior to treatment. (Moderate Recommendation; Evidence Level: Grade B)

11. Prior to offering testosterone therapy, clinicians should measure hemoglobin and hematocrit and inform patients regarding the increased risk of polycythemia. (Strong Recommendation; Evidence Level: Grade A)

12. PSA should be measured in men over 40 years of age prior to commencement of testosterone therapy to exclude a prostate cancer diagnosis. (Clinical Principle)




GUIDELINE STATEMENTS

11. Prior to offering testosterone therapy, clinicians should measure hemoglobin and hematocrit and inform patients regarding the increased risk of polycythemia. (Strong Recommendation; Evidence Level: Grade A)

Polycythemia, sometimes called erythrocytosis, is generally defined as a hematocrit (Hct) >52%. It is categorized into primary (life-long), often related to genetic disorders; and secondary (acquired), which is attributed to polycythemia vera, living at high altitude, hypoxia (e.g., chronic obstructive pulmonary disease, obstructive sleep apnea, tobacco use), paraneoplastic syndromes, and testosterone therapy.187 188

Prior to commencing testosterone therapy, all patients should undergo a baseline measurement of Hb/Hct (Appendix C). If the Hct exceeds 50%, clinicians should consider withholding testosterone therapy until the etiology of the high Hct is explained.187
While on testosterone therapy, a Hct ≥54% warrants intervention. In men with elevated Hct and high on-treatment testosterone levels, dose adjustment should be attempted as first-line management. In men with elevated Hct and low/normal on-treatment testosterone levels, measuring an SHBG level and a free testosterone level using a reliable assay is suggested. If SHBG levels are low/free testosterone levels are high, dose adjustment of the testosterone therapy should be considered. Finally, men with elevated Hct and on treatment low/normal total and free testosterone levels should be referred to a hematologist for further evaluation and possible coordination of phlebotomy.

Androgens have a stimulating effect on erythropoiesis and elevation of Hb/Hct is the most frequent adverse event related to testosterone therapy.189-191 During testosterone therapy, levels of Hb/Hct generally rise for the first six months and then tend to plateau.192, 193

Among 5 randomized, placebo-controlled trials that evaluated Hb and Hct levels in men (mean baseline testosterone <300 ng/dL) using either gel, solution, or IM testosterone therapy for 12 weeks to 1 year, a significant increase in the incidence of elevated Hct was observed in those using testosterone (OR=6.46; CI: 1.86, 22.40) compared to those on placebo.194-201 The calculated odds ratio belies the number of polycythemia events in absolute terms; 19 events in 1,094 patients occurred in the treatment arm as compared to 1 event in 1,093 patients in the placebo group.

While the incidence of polycythemia for one particular modality of testosterone compared to another cannot be determined, trials have indicated that injectable testosterone is associated with the greatest treatment-induced increases in Hb/Hct. In the current meta-analysis of RCTs, long-acting IM testosterone resulted in a mean increase in Hb levels of 1.4 mg/dL compared to 1.6 mg/dL with short-acting IM testosterone, 0.9 mg/dL with transdermal preparations, and 0.7 mg/dL with topical patches.150, 182, 194-196, 201-218 This is likely due to the high and sometimes supra-physiological levels of testosterone that occur in the early days after injection.219 A retrospective comparative series involving 175 men with testosterone deficiency who used different modalities of testosterone therapy reported that 19% of men receiving IM testosterone experienced polycythemia compared to 12.5% with testosterone pellets and 5.4% with gels.220

It is unclear if the risk of polycythemia is greater in men with comorbid disorders that predispose to hypoxia, such as chronic obstructive pulmonary disease or obstructive sleep apnea.188 It is also currently unknown if rates of polycythemia are associated only with short-acting injectable agents or occur with equal frequency when using the longer-acting testosterone undecanoate.221

--------------------------------------------------------------------------------------------------


The use of exogenous T will result in driving up RBCs/hemoglobin/hematocrit within the first month of starting trt and can take up to 9-12 months to reach peak levels.

In many cases, levels can stabilize in due time but many will also struggle with high levels and try to manage with frequent blood donations.

In most cases running too high TT/FT levels will result in high hematocrit.

My reply from another thread:Daily Injection Experiment

Injectable T has been shown to have a greater impact on increasing HCT compared to transdermal T.

3–18% with transdermal administration and up to 44% with injection.

In most cases when using injectable T high supra-physiological peaks post-injection and overall T levels (running too high TT/FT level) will have a big impact on increasing HCT.

Manipulating injection frequency by injecting more frequently using lower doses of T resulting in minimizing the peak--->trough and maintaining more stable levels may lessen the impact on HCT but it is not a given.

As again running very high FT levels will have a stronger impact on driving up HCT.

T formulation, the dose of T, genetics (polymorphism of the AR), age all play a role in the impact a trt protocol will have on blood markers (RBCs/hemoglobin/hematocrit).

Other factors such as sleep apnea, smoking can have a negative impact on hematocrit.

Thanks Madman! Always appreciate your feedback!
 
Again... just to update my current situation. I donated blood on Monday April 5, 2021. Upon checking of my Hemoglobin... it was awfully low. The Red Cross claims it was at 13.8 I quickly made an appointment to my back up Endocrinologist to write some orders for some labs. I needed to know what happened? Why was the Red Cross system off? Or was it really accurate? Didn't make any sense. Here are more newest labs after donating blood.

Labs From March 9, 2021

WBC Count 7.1 K/uL
(Standard Range 4.0 -11.3)
RBC Count 5.71 M/uL (Standard Range 4.30 - 5.90)
Hemoglobin 15.8 g/dL (Standard Range 13.6 -17.6)
*LOWEST HEMOGLOBIN EVER* WOW
Hematocrit 48.3% (Standard Range 40.0 - 50.0%)
*LOWEST HEMATOCRIT EVER* WOW
MCV 84.5 fl (Standard Range 82 - 97)
MCH 27.7 pg (Standard Range 27.3 - 33.4)
MCHC 32.8 g/dL (Standard Range 32 - 36)
RDW 14.4% (Standard Range 11.6 - 14.8)
Platelet Count 269 K/uL (Standard Range 150 - 450)
Mean Platelet Volume 9.2 fL (Standard Range 7.4 - 10.4 fL)

Sodium 141 mmol/L (Standard Range 135 - 145 mmol/L)
Potassium 4.9 mmol/L (Standard Range 3.5 - 5.2 mmol/L)
Chloride 107 mmol/L (Standard Range 98 - 111 mmol/L)
Total CO2 26 mmol/L (Standard Range 20 - 29 mmol/L)
Anion Gap 8 mmol/L (Standard Range 5 - 20 mmol/L)
Glucose 100 mg/dL (Standard Range 60 - 99 mg/dL)
Blood Urea Nitrogen 34 mg/dL (Standard Range 7 - 25 mg/dL)
Creatinine 1.04 mg/dL (Standard Range 0.60 - 1.30 mg/dL)
Glomerular Filtration Rate 88 mL/min/1.73 m2 (Standard Range >60mL)
*Best Filtration Ever On Record* WOW
Calcium 8.8 mg/dL (Standard Range 8.5 - 10.5 mg/dL)
Protein 6.5 g/dL (Standard Range 6.4 - 8.3 g/dL)
Albumin 4.2 gm/dL (Standard Range 3.5 - 5.1 gm/dL)
Bilirubin 0.9 mg/dL (Standard Range 0.3 - 1.2 mg/dL)
AST (SGOT) 33 U/L (Standard Range <35 U/L)
ALT (SGPT) 38 U/L (Standard Range 9 - 47 U/L)
Alkaline Phosphate 60 U/L (Standard Range 33 - 120 U/L)

TSH 4.18 ulU/mL (Standard Range 0.4 - 4.5 ulU/mL)
Free T4 1.00 ng/dL (Standard Range 1.7 - 3.7 ng/dL)

Total Testosterone >1500 (Standard Range 240 - 890 ng/dL)
My doctor only orders the lab in which once past 1500 it doesn't calculate. I couldn't change the order.

PSA Diagnostic 1.0 ng/mL (Standard Range 0.0 - 4.o ng/mL)

It's still mind boggling that I have the lowest RBC, Hemoglobin, Hematocrit since adding Deca and HCG. It blows my mind. I asked my 2nd Endocrinologist what he thought.... and he just said it doesn't make much sense either and I DO NOT ADVISE you taking Deca, HCG on your current protocol. But, I understand you want to live life differently than most people.
This makes sense. So obv ur HGB level from the Red Cross was incorrect. If ur HCT was 48.3 after donating, it was probably around 51 or so before donating. And if ur HGB was 15.8 after donating, it was probably around 17, or slightly over, prior to donating. So I would say that ur HGB and HCT levels are roughly around where they have been in the past, just on the lower end. They’re only better than they’ve been on previous protocols because u tested a few days after donating blood

You are aware that u have clear hypothyroidism correct?

Are u considering dropping ur test dose down a bit after doing less test than usual and feeling better sexuallly?
 
@Stpfan I’m on a similar protocol to urs and have perfect blood pressure. Average 110/60. On my Deca based protocol I also had perfect BP. BP is never an issue regardless of my protocol tbh. I’d be more than happy to give u tips on how to maintain a healthy BP if ur interested. It’s actually pretty simple. I’m at work and just took my BP real quick. That’s currently what it’s at.
 

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Just wanted to update this thread. This is really depressing in a way... I feel like my libido will never return. Takes an awful lot now to feel aroused and I still have ED issues. Viagra at 100mg was working like a charm.... and now... it's not enough. Doesn't seem to be as effective. I can't reach climax at all... without really forcing it... sometimes I can have sex for 45 minutes easily... I'm doing the motions... but not really FEELING anything at all.

This is just an absolute nightmare. I know something is not only hormonal off... but possibly mentally off to. That's just the hand I was dealt in life... that's all you can say. No matter which way I turn... losing a grip on life... like... I was destined to lose out on my sexual abilities. When viagra 100mg doesn't get you ROCK HARD like it was before... it's time to start worrying. Absolutely crazy.
 
Do you ever just decide not to bother climaxing? I'm not suggesting that would be the answer to your problem, but I do think indulging in sex without climax would help your symptoms more than sex with climax. I find my sexual health to be at its best when I keep my orgasms to a minimum.
 
Do you ever just decide not to bother climaxing? I'm not suggesting that would be the answer to your problem, but I do think indulging in sex without climax would help your symptoms more than sex with climax. I find my sexual health to be at its best when I keep my orgasms to a minimum.

I appreciate the response. The issue is... when you do meet a new girl... and have sex for the first time... she finds it very odd you're lasting so long. She believes you aren't into her... then psychologically she thinks there's something wrong with herself. It's a total fu#$ up both ways. You know there's something wrong with you... and now she thinks there's something wrong with her. Then she doesn't want to see you anymore. That's why this situation is getting irritating. Women will be women though... they are pissed off if you climax quickly... lol they get pissed off if you last too long lol I can't win either way. I might just give up... and become a Priest. I don't know? lol
 
I appreciate the response. The issue is... when you do meet a new girl... and have sex for the first time... she finds it very odd you're lasting so long. She believes you aren't into her... then psychologically she thinks there's something wrong with herself. It's a total fu#$ up both ways. You know there's something wrong with you... and now she thinks there's something wrong with her. Then she doesn't want to see you anymore. That's why this situation is getting irritating. Women will be women though... they are pissed off if you climax quickly... lol they get pissed off if you last too long lol I can't win either way. I might just give up... and become a Priest. I don't know? lol
I don't think you need to become a priest, lol, maybe try waiting to have sex until you're married. Much more to relationships than just sex.
 
I don't think you need to become a priest, lol, maybe try waiting to have sex until you're married. Much more to relationships than just sex.

You can't be serious with this post right? From a psychological standpoint... does being married fix someone's ED issues??? Ok then.
 
You can't be serious with this post right? From a psychological standpoint... does being married fix someone's ED issues??? Ok then.
Exactly... Wtf does waiting to be married do. Does your penis know u r getting married and he's waiting to get hard for the married night
 
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