Gents, what do you recommend for a fit, 61 year old guy on TRT for a daily vitamin/supplement regime? I'm currently taking so much stuff, it's getting tiresome keeping up and I sometimes wonder if I'm doing more harm than good. Here is my daily intake minus the quantities.
OMEGA 3 OIL
VIT C,D,E,K,B50
CALCIUM
MAGNESIUM
ZINC
POTASSIUM
ASHWAGANDA
SUPER BETA PROSTATE
DIM
L-CITRILINE
WHEY POWDER
CREATINE
COLLOGEN
TESTOGREENS
I'm also on TC - 120/week, HCG - 1000/week. I feel good, but struggle with getting a good nites sleep and my knees hurt like hell (I have reduced cartilage).
I'd really appreciate hearing what you guys are taking and what works best. Please state your age if you reply.
Thanks!
Steve
Selegeline, 5 mg.
First developed as an anti depressant, deprenyl has clinically proven to work as a cognitive performance booster, sex enhancer, brain protector and much more!
www.antiaging-systems.com
Deprenyl: 50 Years of Life Enhancement and Life Extension
Written by
FARER, Leslie J.
When the drug called deprenyl was first developed in the early 1960’s, the Hungarian doctor who synthesized it could hardly have foreseen its widespread therapeutic potential. Over the past half century, deprenyl has gained recognition not only as a clinically proven antidepressant and Parkinson’s treatment, but also as a mental energizer, physical and cognitive performance booster, generalized mood-lifter, sex enhancer, brain protector, and longevity tonic. The age-retarding and performance-enhancing effects of deprenyl have been clearly demonstrated in animal studies, and humans can benefit from these intriguing findings. The drug is not just beneficial in cases of neurodegenerative diseases; the general healthy but aging population can employ low-dose deprenyl to improve quality of life in their middle to late years, fend off physiological and cognitive decline, boost physical, sexual, and mental vigor, and promote longevity.
Deprenyl Preserves the Essential Neurotransmitter Dopamine
Deprenyl (also known by trade names Selegiline, Jumex, and Eldepryl) exhibits a wide spectrum of pharmacological activity. The first one, for which it is most well-known, is inhibition of an enzyme that breaks down neurotransmitters such as dopamine. (1-5) Let’s take a quick look at the operation of the brain’s circuitry, which depends on the continuous regulation of neurotransmitter levels. In a healthy brain, this achieved by a fine balance between neurotransmitter manufacture from amino acids and breakdown by enzymes. Any disruption of either neurotransmitter synthesis or degradation (or both) can upset the brain’s delicate equilibrium, resulting in mood, psychiatric, or neurological disorders. Here we’re focused on neurotransmitter breakdown, specifically by MAO-B, a member of the monoamine oxidase family of enzymes that degrades dopamine (as well as other brain chemicals such as phenylethylamine (PEA)). Enzymatic degradation is a biochemical pathway necessary for the elimination of used neurotransmitters. MAO-B is essential to brain metabolism, but if its over-activity exceeds the rate of dopamine synthesis, the brain’s dopamine stores become depleted, with disastrous consequences. That’s where deprenyl comes to the rescue: by blocking MAO-B, it prevents the breakdown of dopamine, replenishing the neurotransmitter’s levels in the brain – the crucial mechanism that underlies deprenyl’s success in treating depression and neurodegenerative diseases. Dopamine is central to movement, coordination, sex drive, and cognition, and its production is hindered in these diseases, and also, as we’ll see below, as a consequence of aging.
