5 PROVEN Ways to Fix Low Libido Fast

Pnw0031 · Feb 13, 2025

sammmy said:
Guarana helps a little with dopamine (enjoyment and interest in life, libido, easier to get to orgasm) and it's not due to its caffeine component because pure caffeine doesn't do that.

I'm getting an Amantadine prescription soon. Pramipexole is another option that has been mentioned in the forum.

Just heads up on prami be careful because nausea is very common on it.

I tried it once for sleep and the entire night I couldn’t decide if I wanted to masturbate, gamble or vomit. Didn’t help with sleep much lol!

sammmy · Feb 13, 2025

You should use it at lower doses. You don't have Parkinson's.

DorianGray · Feb 14, 2025

If you have an erection, you have libido. But, one can have libido and not get an erection. That’s the real problem, IMO.

excelnelg · Feb 15, 2025

sammmy said:
Pseudo scientific nonsense.

Libido is controlled by the brain and lack of it indicates a problem in the brain, probably neurotransmitters. No wonder, libido is often restored to even hypersexuality with dopamine agonists. Everything else is secondary to dopamine.

Contemporary impotent medicine has almost zero understanding of the problem. Instead, we are served the usual fluff that it's all in your head and you should drink more water, sleep longer, and go to a psychotherapy LOL

THANK YOU.

Kenp · Feb 15, 2025

sammmy said:
Guarana helps a little with dopamine (enjoyment and interest in life, libido, easier to get to orgasm) and it's not due to its caffeine component because pure caffeine doesn't do that.

I'm getting an Amantadine prescription soon. Pramipexole is another option that has been mentioned in the forum.

Recent PSSD guy who got libido improvements with Prami: Crazy results on new combo - PSSD Forum

"I’m taking like .1 or .15mg 3-4 times a day. Not really any regimen. I sort of start to feel horny within like 30-40 minutes of taking it."

But, dopamine agonists are also risky: Dopamine agonist withdrawal syndrome: implications for patient care - PubMed

Willyt · Feb 18, 2025

DorianGray said:
If you have an erection, you have libido. But, one can have libido and not get an erection. That’s the real problem, IMO.

This is incorrect. Morning wood does not necessarily mean you have sexual desire. You can have one, but not the other. That is the maddening part.

Vince · Feb 18, 2025

Willyt said:
This is incorrect. Morning wood does not necessarily mean you have sexual desire. You can have one, but not the other. That is the maddening part.

That's correct, "morning wood" (also called nocturnal penile tumescence) is not a direct indicator of sexual desire, as it primarily occurs due to physiological processes during sleep, often happening during REM sleep, and is not necessarily linked to sexual thoughts or dreams; essentially, it's a natural function of the body's nervous system while resting, not a sign of active arousal.

sammmy · Feb 18, 2025

Morning wood is due to nerve activation in the genital region in the morning. It means you want to pee or take a dump, pardon my French LOL

DorianGray · Feb 18, 2025

Willyt said:
This is incorrect. Morning wood does not necessarily mean you have sexual desire. You can have one, but not the other. That is the maddening part.

Good point. Similar to effects of PT-141.

Kenp · Feb 19, 2025

DorianGray said:
Good point. Similar to effects of PT-141.

What does PT141 do? Also a dopamine agonist?

DorianGray · Feb 22, 2025

Kenp said:
What does PT141 do? Also a dopamine agonist?

I’m not sure what its mechanism of action is. It produces physiological effects of increased libido/sexual stimulation. Involuntary erections but different than Trimix. Side effects are annoying.

madman · Feb 23, 2025

sammmy said:
Pseudo scientific nonsense.

Libido is controlled by the brain and lack of it indicates a problem in the brain, probably neurotransmitters. No wonder, libido is often restored to even hypersexuality with dopamine agonists. Everything else is secondary to dopamine.

Contemporary impotent medicine has almost zero understanding of the problem. Instead, we are served the usual fluff that it's all in your head and you should drink more water, sleep longer, and go to a psychotherapy LOL

Just dopamine LOL!

Thread 'Sexual Dysfunction with Antidepressants'

Aug 3, 2023

Abstract

Sexual dysfunction is an important unwanted phenomenon of psychotropic medications. This is often underreported and not given due importance, which can often lead to impaired quality of life and less-than-optimal medication compliance. All types of psychotropic medications are linked to sexual side effects. As the underlying mental disorder improves, some of the sexual issues abate, but treatment-related sexual adverse effects may persist over time and are unfortunately underrecognized by clinicians and scarcely investigated in clinical trials. When it...

Key points that need to be stressed here!

* When we discuss the sexual cycle and the role of antidepressants in altering it, it becomes useful to first understand the physiology of the sexual cycle and the neurotransmitters affecting it. Among the neurotransmitters concerned with psychiatry, the following have a role to play in the sexual response cycle as well. In chronological order, desire is promoted by dopamine, norepinephrine (NE), and estrogen, while it is inhibited by serotonin and prolactin. When it comes to arousal, nitric oxide, dopamine, NE, estrogen and testosterone, oxytocin, acetylcholine, and melanocortin enhance it, while serotonin brings down the arousal. In the stage of orgasm, dopamine and nitric oxide help in enhancing the orgasm while serotonin dampens it. When D1 and D5 receptors are stimulated, they help in facilitating sexual arousal in the form of erection in males and receptivity in females. On stimulation of D2 receptors, orgasm comes to fruition, while serotonin reduces sexual activity by blocking dopamine.7