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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Is it true that an elevated hematocrit level on TRT does not require blood donation?
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<blockquote data-quote="Cataceous" data-source="post: 270652" data-attributes="member: 38109"><p>Sure, if you're very unlucky. With such rarity it has little relevance here, and serves as another feeble attempt to rationalize excess dosing.</p><p></p><p style="margin-left: 20px"><em>It affects 1 in 20,000 to 64,000 XY (karyotypically male) births. The condition results in the partial or complete inability of cells to respond to androgens.[2] This unresponsiveness can impair or prevent the development of male genitals, as well as impairing or preventing the development of male secondary sexual characteristics at puberty. It does not significantly impair female genital or sexual development.[3][4] The insensitivity to androgens is therefore clinically significant only when it occurs in genetic males, (i.e. individuals with a Y-chromosome, or more specifically, an SRY gene).[5] Clinical phenotypes in these individuals range from a typical male habitus with mild spermatogenic defect or reduced secondary terminal hair, to a full female habitus, despite the presence of a Y-chromosome.[6]</em></p><p>[<a href="https://en.wikipedia.org/wiki/Androgen_insensitivity_syndrome" target="_blank">R</a>]</p></blockquote><p></p>
[QUOTE="Cataceous, post: 270652, member: 38109"] Sure, if you're very unlucky. With such rarity it has little relevance here, and serves as another feeble attempt to rationalize excess dosing. [INDENT][I]It affects 1 in 20,000 to 64,000 XY (karyotypically male) births. The condition results in the partial or complete inability of cells to respond to androgens.[2] This unresponsiveness can impair or prevent the development of male genitals, as well as impairing or preventing the development of male secondary sexual characteristics at puberty. It does not significantly impair female genital or sexual development.[3][4] The insensitivity to androgens is therefore clinically significant only when it occurs in genetic males, (i.e. individuals with a Y-chromosome, or more specifically, an SRY gene).[5] Clinical phenotypes in these individuals range from a typical male habitus with mild spermatogenic defect or reduced secondary terminal hair, to a full female habitus, despite the presence of a Y-chromosome.[6][/I][/INDENT] [[URL='https://en.wikipedia.org/wiki/Androgen_insensitivity_syndrome']R[/URL]] [/QUOTE]
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Testosterone Replacement, Low T, HCG, & Beyond
Testosterone Side Effect Management
Is it true that an elevated hematocrit level on TRT does not require blood donation?
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